Description

Vitamin E 268 mg

Solgar Vitamin E 268 mg is found naturally in some foods, add to others, and available as a dietary supplement. “Vitamin E” is the collective name for a group of fat-soluble compounds with distinctive antioxidant activities [1].

Naturally occurring vitamin E exists in eight chemical forms (alpha-, beta-, gamma-, and delta-tocopherol and alpha-, beta-, gamma-, and delta-tocotrienol) that have varying levels of biological activity [1]. Alpha- (or α-) tocopherol is the only form that is recognize to meet human requirements

Solgar Vitamin E Benefits

Antioxidants protect cells from the damaging effects of free radicals, which are molecules that contain an unshare electron. Free radicals damage cells and might contribute to the development of cardiovascular disease and cancer [5]. Unshared electrons are highly energetic and react rapidly with oxygen to form reactive oxygen species (ROS).

The body forms ROS endogenously when it converts food to energy. Antioxidants might protect cells from the damaging effects of ROS. The body is also expose to free radicals from environmental exposures. Such as cigarette smoke, air pollution, and ultraviolet radiation from the sun. ROS are part of signaling mechanisms among cells.

Solgar Vitamin E 268 mg is a fat-soluble antioxidant that stops the production of ROS formed when fat undergoes oxidation. Scientists are investigating whether, by limiting free-radical production and possibly through other mechanisms, vitamin E might help prevent or delay the chronic diseases associated with free radicals.

In addition to its activities as an antioxidant, Solgar vitamin E 268 mg is involved in immune function and, as shown primarily by in vitro studies of cells, cell signaling, regulation of gene expression, and other metabolic processes [1].

Alpha-tocopherol inhibits the activity of protein kinase C, an enzyme involved in cell proliferation and differentiation in smooth muscle cells, platelets, and monocytes [6].

Vitamin-E–replete endothelial cells lining the interior surface of blood vessels. They are better able to resist blood-cell components adhering to this surface. Vitamin E also increases the expression of two enzymes that suppress arachidonic acid metabolism.

Thereby increasing the release of prostacyclin from the endothelium.  Which, in turn, dilates blood vessels and inhibits platelet aggregation [